Ontology type: schema:ScholarlyArticle
1992-02
AUTHORS ABSTRACTMuscular fatigue is manifested by a decline in force- or power-generating capacity and may be prominent in both submaximal and maximal contractions. Disturbances in muscle electrolytes play an important role in the development of muscular fatigue. Intense muscular contraction is accompanied by an increased muscle water content, distributed in both intracellular and extra-cellular spaces. This water influx will modify ionic changes in both compartments. Changes in muscle intracellular electrolyte concentrations with intense contraction may be summarised as including decreases in potassium (6 to 20%) and in creatine phosphate (up to 70 to 100%) and increases in lactate (more than 10-fold), sodium (2-fold) and small, variable increases in chloride. The net result of these intracellular ionic concentration changes with exercise will be a reduction in the intracellular strong ion difference, with a consequent marked rise in intracellular hydrogen ion concentration. This intracellular acidosis has been linked with fatigue via impairment of regulatory and contractile protein function, calcium regulation and metabolism. Potassium efflux from the contracting muscle cell dramatically decreases the intracellular to extracellular potassium ratio, leading to depolarisation of sarcolemmal and t-tubular membranes. Surprisingly little research has investigated the effects of intense exercise training on electrolyte regulation and fatigue. Intense sprint training in man attenuates muscular fatigue during short term maximal exercise. This is accompanied by improved potassium homeostasis and possibly, improved regulation of muscular acidosis, both factors which may reduce muscular fatigue. More... »
PAGES134-145
http://scigraph.springernature.com/pub.10.2165/00007256-199213020-00009
DOIhttp://dx.doi.org/10.2165/00007256-199213020-00009
DIMENSIONShttps://app.dimensions.ai/details/publication/pub.1012526655
PUBMEDhttps://www.ncbi.nlm.nih.gov/pubmed/1373245
JSON-LD is the canonical representation for SciGraph data.
TIP: You can open this SciGraph record using an external JSON-LD service: JSON-LD Playground Google SDTT
[
{
"@context": "https://springernature.github.io/scigraph/jsonld/sgcontext.json",
"about": [
{
"id": "http://purl.org/au-research/vocabulary/anzsrc-for/2008/11",
"inDefinedTermSet": "http://purl.org/au-research/vocabulary/anzsrc-for/2008/",
"name": "Medical and Health Sciences",
"type": "DefinedTerm"
},
{
"id": "http://purl.org/au-research/vocabulary/anzsrc-for/2008/1116",
"inDefinedTermSet": "http://purl.org/au-research/vocabulary/anzsrc-for/2008/",
"name": "Medical Physiology",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Acidosis",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Animals",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Chlorides",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Electrolytes",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Exercise",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Fatigue",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Humans",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Ion Channels",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Muscle Contraction",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Muscles",
"type": "DefinedTerm"
},
{
"inDefinedTermSet": "https://www.nlm.nih.gov/mesh/",
"name": "Sodium-Potassium-Exchanging ATPase",
"type": "DefinedTerm"
}
],
"author": [
{
"affiliation": {
"alternateName": "Department of Biological Sciences, Faculty of Health Sciences, The University of Sydney, P.O. Box 170, 2141, Lidcombe, NSW, Australia",
"id": "http://www.grid.ac/institutes/grid.1013.3",
"name": [
"Department of Biological Sciences, Faculty of Health Sciences, The University of Sydney, P.O. Box 170, 2141, Lidcombe, NSW, Australia"
],
"type": "Organization"
},
"familyName": "McKenna",
"givenName": "Michael J.",
"id": "sg:person.01036756372.52",
"sameAs": [
"https://app.dimensions.ai/discover/publication?and_facet_researcher=ur.01036756372.52"
],
"type": "Person"
}
],
"citation": [
{
"id": "sg:pub.10.1007/bf00585248",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1007950830",
"https://doi.org/10.1007/bf00585248"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.1007/bf00580975",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1037310997",
"https://doi.org/10.1007/bf00580975"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.1038/343375a0",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1021716572",
"https://doi.org/10.1038/343375a0"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.1038/293471a0",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1003597152",
"https://doi.org/10.1038/293471a0"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.1038/316736a0",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1015685848",
"https://doi.org/10.1038/316736a0"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.2165/00007256-199111060-00004",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1007382161",
"https://doi.org/10.2165/00007256-199111060-00004"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.1007/bf00635993",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1050263083",
"https://doi.org/10.1007/bf00635993"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.1007/bf00583367",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1014317659",
"https://doi.org/10.1007/bf00583367"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.1007/bf02584013",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1004602665",
"https://doi.org/10.1007/bf02584013"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.1007/bf00656721",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1002395091",
"https://doi.org/10.1007/bf00656721"
],
"type": "CreativeWork"
},
{
"id": "sg:pub.10.1007/978-3-0348-5523-5_41",
"sameAs": [
"https://app.dimensions.ai/details/publication/pub.1008497501",
"https://doi.org/10.1007/978-3-0348-5523-5_41"
],
"type": "CreativeWork"
}
],
"datePublished": "1992-02",
"datePublishedReg": "1992-02-01",
"description": "Muscular fatigue is manifested by a decline in force- or power-generating capacity and may be prominent in both submaximal and maximal contractions. Disturbances in muscle electrolytes play an important role in the development of muscular fatigue. Intense muscular contraction is accompanied by an increased muscle water content, distributed in both intracellular and extra-cellular spaces. This water influx will modify ionic changes in both compartments. Changes in muscle intracellular electrolyte concentrations with intense contraction may be summarised as including decreases in potassium (6 to 20%) and in creatine phosphate (up to 70 to 100%) and increases in lactate (more than 10-fold), sodium (2-fold) and small, variable increases in chloride. The net result of these intracellular ionic concentration changes with exercise will be a reduction in the intracellular strong ion difference, with a consequent marked rise in intracellular hydrogen ion concentration. This intracellular acidosis has been linked with fatigue via impairment of regulatory and contractile protein function, calcium regulation and metabolism. Potassium efflux from the contracting muscle cell dramatically decreases the intracellular to extracellular potassium ratio, leading to depolarisation of sarcolemmal and t-tubular membranes. Surprisingly little research has investigated the effects of intense exercise training on electrolyte regulation and fatigue. Intense sprint training in man attenuates muscular fatigue during short term maximal exercise. This is accompanied by improved potassium homeostasis and possibly, improved regulation of muscular acidosis, both factors which may reduce muscular fatigue.",
"genre": "article",
"id": "sg:pub.10.2165/00007256-199213020-00009",
"isAccessibleForFree": false,
"isPartOf": [
{
"id": "sg:journal.1095007",
"issn": [
"0112-1642",
"1179-2035"
],
"name": "Sports Medicine",
"publisher": "Springer Nature",
"type": "Periodical"
},
{
"issueNumber": "2",
"type": "PublicationIssue"
},
{
"type": "PublicationVolume",
"volumeNumber": "13"
}
],
"keywords": [
"muscular fatigue",
"short-term maximal exercise",
"intense muscular contractions",
"intense exercise training",
"contractile protein function",
"intracellular hydrogen ion concentration",
"contracting muscle cells",
"strong ion difference",
"exercise training",
"maximal contraction",
"maximal exercise",
"muscle electrolytes",
"intracellular electrolyte concentrations",
"intense exercise",
"intracellular acidosis",
"intense contraction",
"potassium ratio",
"marked rise",
"muscle cells",
"potassium efflux",
"potassium homeostasis",
"sprint training",
"muscular contraction",
"electrolyte regulation",
"ion difference",
"creatine phosphate",
"calcium regulation",
"ionic changes",
"acidosis",
"extra-cellular space",
"exercise",
"contraction",
"muscle water content",
"fatigue",
"variable increase",
"ionic concentration changes",
"power-generating capacity",
"sarcolemmal",
"impairment",
"important role",
"regulation",
"men",
"intracellular",
"homeostasis",
"lactate",
"depolarisation",
"role",
"metabolism",
"efflux",
"changes",
"increase",
"training",
"influx",
"cells",
"little research",
"hydrogen ion concentration",
"concentration",
"sodium",
"concentration changes",
"compartments",
"net result",
"factors",
"differences",
"decline",
"decrease",
"potassium",
"reduction",
"effect",
"disturbances",
"protein function",
"function",
"membrane",
"development",
"ratio",
"rise",
"results",
"tubular membranes",
"capacity",
"phosphate",
"electrolyte concentration",
"research",
"ion concentration",
"chloride",
"water influx",
"content",
"process",
"force",
"water content",
"ionic processes",
"space",
"electrolyte"
],
"name": "The Roles of Ionic Processes in Muscular Fatigue During Intense Exercise",
"pagination": "134-145",
"productId": [
{
"name": "dimensions_id",
"type": "PropertyValue",
"value": [
"pub.1012526655"
]
},
{
"name": "doi",
"type": "PropertyValue",
"value": [
"10.2165/00007256-199213020-00009"
]
},
{
"name": "pubmed_id",
"type": "PropertyValue",
"value": [
"1373245"
]
}
],
"sameAs": [
"https://doi.org/10.2165/00007256-199213020-00009",
"https://app.dimensions.ai/details/publication/pub.1012526655"
],
"sdDataset": "articles",
"sdDatePublished": "2022-08-04T16:51",
"sdLicense": "https://scigraph.springernature.com/explorer/license/",
"sdPublisher": {
"name": "Springer Nature - SN SciGraph project",
"type": "Organization"
},
"sdSource": "s3://com-springernature-scigraph/baseset/20220804/entities/gbq_results/article/article_231.jsonl",
"type": "ScholarlyArticle",
"url": "https://doi.org/10.2165/00007256-199213020-00009"
}
]
Download the RDF metadata as: json-ld nt turtle xml License info
JSON-LD is a popular format for linked data which is fully compatible with JSON.
curl -H 'Accept: application/ld+json' 'https://scigraph.springernature.com/pub.10.2165/00007256-199213020-00009'
N-Triples is a line-based linked data format ideal for batch operations.
curl -H 'Accept: application/n-triples' 'https://scigraph.springernature.com/pub.10.2165/00007256-199213020-00009'
Turtle is a human-readable linked data format.
curl -H 'Accept: text/turtle' 'https://scigraph.springernature.com/pub.10.2165/00007256-199213020-00009'
RDF/XML is a standard XML format for linked data.
curl -H 'Accept: application/rdf+xml' 'https://scigraph.springernature.com/pub.10.2165/00007256-199213020-00009'
This table displays all metadata directly associated to this object as RDF triples.
240 TRIPLES
21 PREDICATES
139 URIs
120 LITERALS
18 BLANK NODES