The Helicobacter felis model of adoptive transfer gastritis View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2005-11

AUTHORS

Vance J. McCracken, Steven M. Martin, Robin G. Lorenz

ABSTRACT

The bacterium Helicobacter pylori is a major human pathogen and the principal cause of acute and chronic gastritis, gastric and duodenal ulcer disease, and gastric adenocarcinoma. Infection with gastric Helicobacter results in an early infiltration of neutrophils, monocytes, and natural killer cells, followed by an influx of T cells and plasma cells. Although the critical components of this gastric infiltrate that lead to disease are unclear, the Helicobacter felis-infected mouse and other mouse models of Helicobacter-associated gastritis have demonstrated the critical nature of adaptive immunity in the development of gastric epithelial pathology. To further investigate the role of adaptive immunity in this disease, adoptive transfer models of disease have also been utilized. These models clearly demonstrate that it is the host CD4+ T lymphocyte response that is crucial for the development of Helicobacter-associated gastric epithelial changes. More... »

PAGES

183-194

References to SciGraph publications

  • 1998-10. HELICOBACTER COLONIZATION OF THE BILIARY TREE: COMMENSAL, PATHOGEN, OR SPURIOUS FINDING? in THE AMERICAN JOURNAL OF GASTROENTEROLOGY
  • 2005-03-10. Helicobacter pylori VacA, a paradigm for toxin multifunctionality in NATURE REVIEWS MICROBIOLOGY
  • 1999. Animal Models of Helicobacter Gastritis in GASTRODUODENAL DISEASE AND HELICOBACTER PYLORI
  • 2001-06. Living dangerously: how Helicobacter pylori survives in the human stomach in NATURE REVIEWS MOLECULAR CELL BIOLOGY
  • 2002-05-13. In vivo developmental stages in murine natural killer cell maturation in NATURE IMMUNOLOGY
  • Identifiers

    URI

    http://scigraph.springernature.com/pub.10.1385/ir:33:2:183

    DOI

    http://dx.doi.org/10.1385/ir:33:2:183

    DIMENSIONS

    https://app.dimensions.ai/details/publication/pub.1020737469

    PUBMED

    https://www.ncbi.nlm.nih.gov/pubmed/16234583


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