Bench-to-bedside review: Endothelial cell dysfunction in severe sepsis: a role in organ dysfunction? View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

2003-04

AUTHORS

Benoît Vallet

ABSTRACT

During the past decade a unifying hypothesis has been developed to explain the vascular changes that occur in septic shock on the basis of the effect of inflammatory mediators on the vascular endothelium. The vascular endothelium plays a central role in the control of microvascular flow, and it has been proposed that widespread vascular endothelial activation, dysfunction and eventually injury occurs in septic shock, ultimately resulting in multiorgan failure. This has been characterized in various models of experimental septic shock. Now, direct and indirect evidence for endothelial cell alteration in humans during septic shock is emerging. The present review details recently published literature on this rapidly evolving topic. More... »

PAGES

130

Identifiers

URI

http://scigraph.springernature.com/pub.10.1186/cc1864

DOI

http://dx.doi.org/10.1186/cc1864

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1029448751

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/12720559


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