Donor HSCs with a preexisting ASXL1-mutation led to the development of FLT3-ITD positive AML in the donor and FLT3-ITD negative ... View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


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URI

http://scigraph.springernature.com/pub.10.1038/s41409-017-0046-8

DOI

http://dx.doi.org/10.1038/s41409-017-0046-8

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https://app.dimensions.ai/details/publication/pub.1100248962

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/29330388


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