Psoriasis View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

2016-11-24

AUTHORS

Jacqueline E. Greb, Ari M. Goldminz, James T. Elder, Mark G. Lebwohl, Dafna D. Gladman, Jashin J. Wu, Nehal N. Mehta, Andrew Y. Finlay, Alice B. Gottlieb

ABSTRACT

Psoriasis is a chronic, immune-mediated disorder with cutaneous and systemic manifestations and substantial negative effects on patient quality of life. Psoriasis has a strong, albeit polygenic, genetic basis. Whereas approximately half of the accountable genetic effect of psoriasis maps to the major histocompatibility complex, >70 other loci have been identified, many of which implicate nuclear factor-κB, interferon signalling and the IL-23-IL-23 receptor axis. Psoriasis pathophysiology is characterized by abnormal keratinocyte proliferation and immune cell infiltration in the dermis and epidermis involving the innate and adaptive immune systems, with important roles for dendritic cells and T cells, among other cells. Frequent comorbidities are rheumatological and cardiovascular in nature, in particular, psoriatic arthritis. Current treatments for psoriasis include topical agents, photo-based therapies, traditional systemic drugs and biologic agents. Treatments can be used in combination or as monotherapy. Biologic therapies that target specific disease mediators have become a mainstay in the treatment of moderate-to-severe disease, whereas advances in the treatment of mild-to-moderate disease have been limited. More... »

PAGES

nrdp201682

Identifiers

URI

http://scigraph.springernature.com/pub.10.1038/nrdp.2016.82

DOI

http://dx.doi.org/10.1038/nrdp.2016.82

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1016768384

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/27883001


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