Novel therapeutic approaches for heart failure by normalizing calcium cycling View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2004-07

AUTHORS

Xander H. T. Wehrens, Andrew R. Marks

ABSTRACT

Congestive heart failure is the leading cause of death in the Western world. Abnormal intracellular calcium (Ca2+) handling is central to the pathogenesis of heart failure because it contributes to a decrease in ventricular contractile function. Chronic hyperactivity of the sympathetic nervous system causes increased phosphorylation of the ryanodine receptor intracellular Ca2+-release channel, a key Ca2+-handling protein in the heart, by protein kinase A. Alteration of the structure and function of ryanodine receptors contributes to defective intracellular Ca2+ handling and an increased propensity for cardiac arrhythmias in failing hearts. Novel therapeutic strategies are now being evaluated to specifically correct defective Ca2+-handling in heart failure. More... »

PAGES

565

References to SciGraph publications

Identifiers

URI

http://scigraph.springernature.com/pub.10.1038/nrd1440

DOI

http://dx.doi.org/10.1038/nrd1440

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1052981264

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/15232578


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