Pushing the limits of targeted therapy in chronic myeloid leukaemia View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2012-08

AUTHORS

Thomas O'Hare, Matthew S. Zabriskie, Anna M. Eiring, Michael W. Deininger

ABSTRACT

Tyrosine kinase inhibitor (TKI) therapy targeting the BCR-ABL1 kinase is effective against chronic myeloid leukaemia (CML), but is not curative for most patients. Minimal residual disease (MRD) is thought to reside in TKI-insensitive leukaemia stem cells (LSCs) that are not fully addicted to BCR-ABL1. Recent conceptual advances in both CML biology and therapeutic intervention have increased the potential for the elimination of CML cells, including LSCs, through simultaneous inhibition of BCR-ABL1 and other newly identified, crucial targets. More... »

PAGES

513

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  • Identifiers

    URI

    http://scigraph.springernature.com/pub.10.1038/nrc3317

    DOI

    http://dx.doi.org/10.1038/nrc3317

    DIMENSIONS

    https://app.dimensions.ai/details/publication/pub.1051689689

    PUBMED

    https://www.ncbi.nlm.nih.gov/pubmed/22825216


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