EphB receptors regulate dendritic spine development via intersectin, Cdc42 and N-WASP View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2002-11

AUTHORS

Fumitoshi Irie, Yu Yamaguchi

ABSTRACT

Dendritic spines show structural plasticity during development and in response to synaptic activity1,2,3. We previously found that EphB2 receptor tyrosine kinase, a receptor for B-ephrin ligands, is functionally involved in spine morphogenesis4, but the downstream signaling mechanisms are not known. Here we show that EphB2 physically associates with the guanine nucleotide exchange factor (GEF) intersectin and activates its GEF activity in cooperation with neural Wiskott-Aldrich syndrome protein (N-WASP), which in turn activates the Rho-family GTPase Cdc42 and spine morphogenesis. More... »

PAGES

1117

Identifiers

URI

http://scigraph.springernature.com/pub.10.1038/nn964

DOI

http://dx.doi.org/10.1038/nn964

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1032251157

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/12389031


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