Cyclophilin A enhances vascular oxidative stress and the development of angiotensin II–induced aortic aneurysms View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

2009-05-10

AUTHORS

Kimio Satoh, Patrizia Nigro, Tetsuya Matoba, Michael R O'Dell, Zhaoqiang Cui, Xi Shi, Amy Mohan, Chen Yan, Jun-ichi Abe, Karl A Illig, Bradford C Berk

ABSTRACT

The pathogenesis of aortic aneurysms involves inflammatory cell recruitment and increased levels of reactive oxygen species and matrix metalloproteases. Kimio Satoh et al. now mechanistically link the protein cyclophilin A—expressed in vascular smooth muscle cells—to these known mediators of aortic aneurysm formation and provide evidence in both mice and humans for the importance of cyclophilin A in aortic aneurysm formation. More... »

PAGES

649-656

Identifiers

URI

http://scigraph.springernature.com/pub.10.1038/nm.1958

DOI

http://dx.doi.org/10.1038/nm.1958

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1045339338

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/19430489


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