Autoimmunity provoked by infection: how good is the case for T cell epitope mimicry? View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2001-09

AUTHORS

Christophe Benoist, Diane Mathis

ABSTRACT

Autoimmune diseases remain one of the mysteries that perplex immunologists. What makes the immune system, which has evolved to protect an organism from foreign invaders, turn on the organism itself? A popular answer to this question involves the lymphoid network's primordial function: autoimmunity is a by-product of the immune response to microbial infection. For decades there have been tantalizing associations between infectious agents and autoimmunity: β-hemolytic streptococci and rheumatic fever; B3 Coxsackieviruses and myocarditis; Trypanosoma cruzi and Chagas' disease; diverse viruses and multiple sclerosis; Borrelia burgdorfii and Lyme arthritis; and B4 Coxsackievirus, cytomegalovirus or rubella and type 1 diabetes, to name the most frequently cited examples1. In addition, animal models have provided direct evidence that infection with a particular microbe can incite a particular autoimmune disease2. Nonetheless, many of the associations appear less than convincing and, even for those that seem to be on solid footing, there is no real understanding of the underlying mechanism(s). More... »

PAGES

797-801

Identifiers

URI

http://scigraph.springernature.com/pub.10.1038/ni0901-797

DOI

http://dx.doi.org/10.1038/ni0901-797

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1051452926

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/11526389


Indexing Status Check whether this publication has been indexed by Scopus and Web Of Science using the SN Indexing Status Tool
Incoming Citations Browse incoming citations for this publication using opencitations.net

JSON-LD is the canonical representation for SciGraph data.

TIP: You can open this SciGraph record using an external JSON-LD service: JSON-LD Playground Google SDTT

[
  {
    "@context": "https://springernature.github.io/scigraph/jsonld/sgcontext.json", 
    "about": [
      {
        "id": "http://purl.org/au-research/vocabulary/anzsrc-for/2008/11", 
        "inDefinedTermSet": "http://purl.org/au-research/vocabulary/anzsrc-for/2008/", 
        "name": "Medical and Health Sciences", 
        "type": "DefinedTerm"
      }, 
      {
        "id": "http://purl.org/au-research/vocabulary/anzsrc-for/2008/1107", 
        "inDefinedTermSet": "http://purl.org/au-research/vocabulary/anzsrc-for/2008/", 
        "name": "Immunology", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Antigens, Surface", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Autoantigens", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Autoimmune Diseases", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Autoimmunity", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Bacterial Outer Membrane Proteins", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Bacterial Vaccines", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Capsid", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Capsid Proteins", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Epitopes, T-Lymphocyte", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Eye Proteins", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Humans", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Keratitis, Herpetic", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Lipoproteins", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Lyme Disease", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Lyme Disease Vaccines", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Lymphocyte Function-Associated Antigen-1", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Models, Immunological", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Molecular Mimicry", 
        "type": "DefinedTerm"
      }, 
      {
        "inDefinedTermSet": "https://www.nlm.nih.gov/mesh/", 
        "name": "Viral Proteins", 
        "type": "DefinedTerm"
      }
    ], 
    "author": [
      {
        "affiliation": {
          "alternateName": "Department of Medicine, Section on Immunology and Immunogenetics, Joslin Diabetes Center, Brigham and Women's Hospital, Harvard Medical School, One Joslin Place, Boston, MA, USA", 
          "id": "http://www.grid.ac/institutes/grid.62560.37", 
          "name": [
            "Department of Medicine, Section on Immunology and Immunogenetics, Joslin Diabetes Center, Brigham and Women's Hospital, Harvard Medical School, One Joslin Place, Boston, MA, USA"
          ], 
          "type": "Organization"
        }, 
        "familyName": "Benoist", 
        "givenName": "Christophe", 
        "id": "sg:person.01231653404.27", 
        "sameAs": [
          "https://app.dimensions.ai/discover/publication?and_facet_researcher=ur.01231653404.27"
        ], 
        "type": "Person"
      }, 
      {
        "affiliation": {
          "alternateName": "Department of Medicine, Section on Immunology and Immunogenetics, Joslin Diabetes Center, Brigham and Women's Hospital, Harvard Medical School, One Joslin Place, Boston, MA, USA", 
          "id": "http://www.grid.ac/institutes/grid.62560.37", 
          "name": [
            "Department of Medicine, Section on Immunology and Immunogenetics, Joslin Diabetes Center, Brigham and Women's Hospital, Harvard Medical School, One Joslin Place, Boston, MA, USA"
          ], 
          "type": "Organization"
        }, 
        "familyName": "Mathis", 
        "givenName": "Diane", 
        "id": "sg:person.0676326063.78", 
        "sameAs": [
          "https://app.dimensions.ai/discover/publication?and_facet_researcher=ur.0676326063.78"
        ], 
        "type": "Person"
      }
    ], 
    "citation": [
      {
        "id": "sg:pub.10.1007/pl00000717", 
        "sameAs": [
          "https://app.dimensions.ai/details/publication/pub.1051823953", 
          "https://doi.org/10.1007/pl00000717"
        ], 
        "type": "CreativeWork"
      }, 
      {
        "id": "sg:pub.10.1007/pl00000641", 
        "sameAs": [
          "https://app.dimensions.ai/details/publication/pub.1041845045", 
          "https://doi.org/10.1007/pl00000641"
        ], 
        "type": "CreativeWork"
      }, 
      {
        "id": "sg:pub.10.1038/70946", 
        "sameAs": [
          "https://app.dimensions.ai/details/publication/pub.1048917118", 
          "https://doi.org/10.1038/70946"
        ], 
        "type": "CreativeWork"
      }, 
      {
        "id": "sg:pub.10.1038/376431a0", 
        "sameAs": [
          "https://app.dimensions.ai/details/publication/pub.1003935766", 
          "https://doi.org/10.1038/376431a0"
        ], 
        "type": "CreativeWork"
      }, 
      {
        "id": "sg:pub.10.1007/pl00000716", 
        "sameAs": [
          "https://app.dimensions.ai/details/publication/pub.1031417794", 
          "https://doi.org/10.1007/pl00000716"
        ], 
        "type": "CreativeWork"
      }
    ], 
    "datePublished": "2001-09", 
    "datePublishedReg": "2001-09-01", 
    "description": "Autoimmune diseases remain one of the mysteries that perplex immunologists. What makes the immune system, which has evolved to protect an organism from foreign invaders, turn on the organism itself? A popular answer to this question involves the lymphoid network's primordial function: autoimmunity is a by-product of the immune response to microbial infection. For decades there have been tantalizing associations between infectious agents and autoimmunity: \u03b2-hemolytic streptococci and rheumatic fever; B3 Coxsackieviruses and myocarditis; Trypanosoma cruzi and Chagas' disease; diverse viruses and multiple sclerosis; Borrelia burgdorfii and Lyme arthritis; and B4 Coxsackievirus, cytomegalovirus or rubella and type 1 diabetes, to name the most frequently cited examples1. In addition, animal models have provided direct evidence that infection with a particular microbe can incite a particular autoimmune disease2. Nonetheless, many of the associations appear less than convincing and, even for those that seem to be on solid footing, there is no real understanding of the underlying mechanism(s).", 
    "genre": "article", 
    "id": "sg:pub.10.1038/ni0901-797", 
    "inLanguage": "en", 
    "isAccessibleForFree": false, 
    "isPartOf": [
      {
        "id": "sg:journal.1022778", 
        "issn": [
          "1529-2908", 
          "1529-2916"
        ], 
        "name": "Nature Immunology", 
        "publisher": "Springer Nature", 
        "type": "Periodical"
      }, 
      {
        "issueNumber": "9", 
        "type": "PublicationIssue"
      }, 
      {
        "type": "PublicationVolume", 
        "volumeNumber": "2"
      }
    ], 
    "keywords": [
      "cell epitope mimicry", 
      "type 1 diabetes", 
      "rheumatic fever", 
      "Lyme arthritis", 
      "epitope mimicry", 
      "multiple sclerosis", 
      "autoimmune diseases", 
      "immune response", 
      "hemolytic streptococci", 
      "animal models", 
      "infectious agents", 
      "immune system", 
      "autoimmunity", 
      "microbial infections", 
      "infection", 
      "disease", 
      "Chagas disease", 
      "foreign invaders", 
      "diverse viruses", 
      "particular microbes", 
      "Trypanosoma cruzi", 
      "coxsackievirus", 
      "association", 
      "myocarditis", 
      "arthritis", 
      "sclerosis", 
      "fever", 
      "rubella", 
      "diabetes", 
      "cytomegalovirus", 
      "disease2", 
      "direct evidence", 
      "immunologists", 
      "streptococci", 
      "virus", 
      "Borrelia", 
      "cruzi", 
      "agents", 
      "response", 
      "evidence", 
      "primordial function", 
      "cases", 
      "mimicry", 
      "organisms", 
      "function", 
      "addition", 
      "popular answer", 
      "decades", 
      "microbes", 
      "understanding", 
      "questions", 
      "real understanding", 
      "model", 
      "mystery", 
      "answers", 
      "products", 
      "solid footing", 
      "system", 
      "invaders", 
      "footing", 
      "examples1", 
      "perplex immunologists", 
      "lymphoid network's primordial function", 
      "network's primordial function", 
      "B3 Coxsackieviruses", 
      "B4 Coxsackievirus", 
      "particular autoimmune disease2", 
      "autoimmune disease2"
    ], 
    "name": "Autoimmunity provoked by infection: how good is the case for T cell epitope mimicry?", 
    "pagination": "797-801", 
    "productId": [
      {
        "name": "dimensions_id", 
        "type": "PropertyValue", 
        "value": [
          "pub.1051452926"
        ]
      }, 
      {
        "name": "doi", 
        "type": "PropertyValue", 
        "value": [
          "10.1038/ni0901-797"
        ]
      }, 
      {
        "name": "pubmed_id", 
        "type": "PropertyValue", 
        "value": [
          "11526389"
        ]
      }
    ], 
    "sameAs": [
      "https://doi.org/10.1038/ni0901-797", 
      "https://app.dimensions.ai/details/publication/pub.1051452926"
    ], 
    "sdDataset": "articles", 
    "sdDatePublished": "2021-11-01T18:05", 
    "sdLicense": "https://scigraph.springernature.com/explorer/license/", 
    "sdPublisher": {
      "name": "Springer Nature - SN SciGraph project", 
      "type": "Organization"
    }, 
    "sdSource": "s3://com-springernature-scigraph/baseset/20211101/entities/gbq_results/article/article_339.jsonl", 
    "type": "ScholarlyArticle", 
    "url": "https://doi.org/10.1038/ni0901-797"
  }
]
 

Download the RDF metadata as:  json-ld nt turtle xml License info

HOW TO GET THIS DATA PROGRAMMATICALLY:

JSON-LD is a popular format for linked data which is fully compatible with JSON.

curl -H 'Accept: application/ld+json' 'https://scigraph.springernature.com/pub.10.1038/ni0901-797'

N-Triples is a line-based linked data format ideal for batch operations.

curl -H 'Accept: application/n-triples' 'https://scigraph.springernature.com/pub.10.1038/ni0901-797'

Turtle is a human-readable linked data format.

curl -H 'Accept: text/turtle' 'https://scigraph.springernature.com/pub.10.1038/ni0901-797'

RDF/XML is a standard XML format for linked data.

curl -H 'Accept: application/rdf+xml' 'https://scigraph.springernature.com/pub.10.1038/ni0901-797'


 

This table displays all metadata directly associated to this object as RDF triples.

233 TRIPLES      22 PREDICATES      119 URIs      106 LITERALS      26 BLANK NODES

Subject Predicate Object
1 sg:pub.10.1038/ni0901-797 schema:about N05beeaea775c4df7b66a3b2f49ae3009
2 N08ce28ebd418473a9d3b13c7dcdf21a8
3 N1d4359a8eb0f4b2cba92926a34cfdd3d
4 N203e6540bb514dd0b8fd13e45bb4c7b5
5 N424ab6041bf34a7499e186093b388573
6 N43e55802935e41429465cdbe8a42dcd6
7 N493f8edbe9804b439ebb985942b4e0ad
8 N5af96066eba64ba3a0791b72e0545cc8
9 N5eea300d917f41cfb7e765a3f58dfa09
10 N79c203462bdd4ece9e5672b64ed353f2
11 N7c9450534c2742a0ba172c7a51995fd4
12 N8ca1c5139a8b44c4b7ff801b825ce232
13 N8fec326288ea432799767cc00aed01bc
14 N9c8d5d8710e440af837b3cb2c08255e7
15 Na509a13ee97a46188c23dfa8ce309775
16 Na96b639456884a0a8ec644c65082ce0c
17 Nb2391d05eb064ff3b5a07ef6ad2a27b8
18 Nc53e547ce88543ccafba5fa3f383e780
19 Nfa7820b5b3ba4236bdcc2870143b000c
20 anzsrc-for:11
21 anzsrc-for:1107
22 schema:author Nbed4f5b7fa66458382ade41c1569818f
23 schema:citation sg:pub.10.1007/pl00000641
24 sg:pub.10.1007/pl00000716
25 sg:pub.10.1007/pl00000717
26 sg:pub.10.1038/376431a0
27 sg:pub.10.1038/70946
28 schema:datePublished 2001-09
29 schema:datePublishedReg 2001-09-01
30 schema:description Autoimmune diseases remain one of the mysteries that perplex immunologists. What makes the immune system, which has evolved to protect an organism from foreign invaders, turn on the organism itself? A popular answer to this question involves the lymphoid network's primordial function: autoimmunity is a by-product of the immune response to microbial infection. For decades there have been tantalizing associations between infectious agents and autoimmunity: β-hemolytic streptococci and rheumatic fever; B3 Coxsackieviruses and myocarditis; Trypanosoma cruzi and Chagas' disease; diverse viruses and multiple sclerosis; Borrelia burgdorfii and Lyme arthritis; and B4 Coxsackievirus, cytomegalovirus or rubella and type 1 diabetes, to name the most frequently cited examples1. In addition, animal models have provided direct evidence that infection with a particular microbe can incite a particular autoimmune disease2. Nonetheless, many of the associations appear less than convincing and, even for those that seem to be on solid footing, there is no real understanding of the underlying mechanism(s).
31 schema:genre article
32 schema:inLanguage en
33 schema:isAccessibleForFree false
34 schema:isPartOf N02a39feb92744b0a9b37aa7f2079def2
35 Ne2d39c578ef54e3ea88300b5577eb971
36 sg:journal.1022778
37 schema:keywords B3 Coxsackieviruses
38 B4 Coxsackievirus
39 Borrelia
40 Chagas disease
41 Lyme arthritis
42 Trypanosoma cruzi
43 addition
44 agents
45 animal models
46 answers
47 arthritis
48 association
49 autoimmune disease2
50 autoimmune diseases
51 autoimmunity
52 cases
53 cell epitope mimicry
54 coxsackievirus
55 cruzi
56 cytomegalovirus
57 decades
58 diabetes
59 direct evidence
60 disease
61 disease2
62 diverse viruses
63 epitope mimicry
64 evidence
65 examples1
66 fever
67 footing
68 foreign invaders
69 function
70 hemolytic streptococci
71 immune response
72 immune system
73 immunologists
74 infection
75 infectious agents
76 invaders
77 lymphoid network's primordial function
78 microbes
79 microbial infections
80 mimicry
81 model
82 multiple sclerosis
83 myocarditis
84 mystery
85 network's primordial function
86 organisms
87 particular autoimmune disease2
88 particular microbes
89 perplex immunologists
90 popular answer
91 primordial function
92 products
93 questions
94 real understanding
95 response
96 rheumatic fever
97 rubella
98 sclerosis
99 solid footing
100 streptococci
101 system
102 type 1 diabetes
103 understanding
104 virus
105 schema:name Autoimmunity provoked by infection: how good is the case for T cell epitope mimicry?
106 schema:pagination 797-801
107 schema:productId N7a0fd390f14947908e501f7410f24f87
108 Nf98c7286a6334ce3a9309926749c7711
109 Nfe2f0d8c2cd44f9d9beb3a15d5653c68
110 schema:sameAs https://app.dimensions.ai/details/publication/pub.1051452926
111 https://doi.org/10.1038/ni0901-797
112 schema:sdDatePublished 2021-11-01T18:05
113 schema:sdLicense https://scigraph.springernature.com/explorer/license/
114 schema:sdPublisher N1a748c5f5c9b4d39931747ad971e1374
115 schema:url https://doi.org/10.1038/ni0901-797
116 sgo:license sg:explorer/license/
117 sgo:sdDataset articles
118 rdf:type schema:ScholarlyArticle
119 N02a39feb92744b0a9b37aa7f2079def2 schema:issueNumber 9
120 rdf:type schema:PublicationIssue
121 N05beeaea775c4df7b66a3b2f49ae3009 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
122 schema:name Capsid Proteins
123 rdf:type schema:DefinedTerm
124 N08ce28ebd418473a9d3b13c7dcdf21a8 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
125 schema:name Bacterial Vaccines
126 rdf:type schema:DefinedTerm
127 N1a748c5f5c9b4d39931747ad971e1374 schema:name Springer Nature - SN SciGraph project
128 rdf:type schema:Organization
129 N1d4359a8eb0f4b2cba92926a34cfdd3d schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
130 schema:name Viral Proteins
131 rdf:type schema:DefinedTerm
132 N203e6540bb514dd0b8fd13e45bb4c7b5 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
133 schema:name Autoimmune Diseases
134 rdf:type schema:DefinedTerm
135 N424ab6041bf34a7499e186093b388573 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
136 schema:name Lymphocyte Function-Associated Antigen-1
137 rdf:type schema:DefinedTerm
138 N43e55802935e41429465cdbe8a42dcd6 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
139 schema:name Capsid
140 rdf:type schema:DefinedTerm
141 N4454f10fee28433bb847e7703595f915 rdf:first sg:person.0676326063.78
142 rdf:rest rdf:nil
143 N493f8edbe9804b439ebb985942b4e0ad schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
144 schema:name Lyme Disease Vaccines
145 rdf:type schema:DefinedTerm
146 N5af96066eba64ba3a0791b72e0545cc8 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
147 schema:name Autoantigens
148 rdf:type schema:DefinedTerm
149 N5eea300d917f41cfb7e765a3f58dfa09 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
150 schema:name Lipoproteins
151 rdf:type schema:DefinedTerm
152 N79c203462bdd4ece9e5672b64ed353f2 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
153 schema:name Humans
154 rdf:type schema:DefinedTerm
155 N7a0fd390f14947908e501f7410f24f87 schema:name pubmed_id
156 schema:value 11526389
157 rdf:type schema:PropertyValue
158 N7c9450534c2742a0ba172c7a51995fd4 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
159 schema:name Autoimmunity
160 rdf:type schema:DefinedTerm
161 N8ca1c5139a8b44c4b7ff801b825ce232 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
162 schema:name Antigens, Surface
163 rdf:type schema:DefinedTerm
164 N8fec326288ea432799767cc00aed01bc schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
165 schema:name Keratitis, Herpetic
166 rdf:type schema:DefinedTerm
167 N9c8d5d8710e440af837b3cb2c08255e7 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
168 schema:name Lyme Disease
169 rdf:type schema:DefinedTerm
170 Na509a13ee97a46188c23dfa8ce309775 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
171 schema:name Eye Proteins
172 rdf:type schema:DefinedTerm
173 Na96b639456884a0a8ec644c65082ce0c schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
174 schema:name Epitopes, T-Lymphocyte
175 rdf:type schema:DefinedTerm
176 Nb2391d05eb064ff3b5a07ef6ad2a27b8 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
177 schema:name Bacterial Outer Membrane Proteins
178 rdf:type schema:DefinedTerm
179 Nbed4f5b7fa66458382ade41c1569818f rdf:first sg:person.01231653404.27
180 rdf:rest N4454f10fee28433bb847e7703595f915
181 Nc53e547ce88543ccafba5fa3f383e780 schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
182 schema:name Molecular Mimicry
183 rdf:type schema:DefinedTerm
184 Ne2d39c578ef54e3ea88300b5577eb971 schema:volumeNumber 2
185 rdf:type schema:PublicationVolume
186 Nf98c7286a6334ce3a9309926749c7711 schema:name dimensions_id
187 schema:value pub.1051452926
188 rdf:type schema:PropertyValue
189 Nfa7820b5b3ba4236bdcc2870143b000c schema:inDefinedTermSet https://www.nlm.nih.gov/mesh/
190 schema:name Models, Immunological
191 rdf:type schema:DefinedTerm
192 Nfe2f0d8c2cd44f9d9beb3a15d5653c68 schema:name doi
193 schema:value 10.1038/ni0901-797
194 rdf:type schema:PropertyValue
195 anzsrc-for:11 schema:inDefinedTermSet anzsrc-for:
196 schema:name Medical and Health Sciences
197 rdf:type schema:DefinedTerm
198 anzsrc-for:1107 schema:inDefinedTermSet anzsrc-for:
199 schema:name Immunology
200 rdf:type schema:DefinedTerm
201 sg:journal.1022778 schema:issn 1529-2908
202 1529-2916
203 schema:name Nature Immunology
204 schema:publisher Springer Nature
205 rdf:type schema:Periodical
206 sg:person.01231653404.27 schema:affiliation grid-institutes:grid.62560.37
207 schema:familyName Benoist
208 schema:givenName Christophe
209 schema:sameAs https://app.dimensions.ai/discover/publication?and_facet_researcher=ur.01231653404.27
210 rdf:type schema:Person
211 sg:person.0676326063.78 schema:affiliation grid-institutes:grid.62560.37
212 schema:familyName Mathis
213 schema:givenName Diane
214 schema:sameAs https://app.dimensions.ai/discover/publication?and_facet_researcher=ur.0676326063.78
215 rdf:type schema:Person
216 sg:pub.10.1007/pl00000641 schema:sameAs https://app.dimensions.ai/details/publication/pub.1041845045
217 https://doi.org/10.1007/pl00000641
218 rdf:type schema:CreativeWork
219 sg:pub.10.1007/pl00000716 schema:sameAs https://app.dimensions.ai/details/publication/pub.1031417794
220 https://doi.org/10.1007/pl00000716
221 rdf:type schema:CreativeWork
222 sg:pub.10.1007/pl00000717 schema:sameAs https://app.dimensions.ai/details/publication/pub.1051823953
223 https://doi.org/10.1007/pl00000717
224 rdf:type schema:CreativeWork
225 sg:pub.10.1038/376431a0 schema:sameAs https://app.dimensions.ai/details/publication/pub.1003935766
226 https://doi.org/10.1038/376431a0
227 rdf:type schema:CreativeWork
228 sg:pub.10.1038/70946 schema:sameAs https://app.dimensions.ai/details/publication/pub.1048917118
229 https://doi.org/10.1038/70946
230 rdf:type schema:CreativeWork
231 grid-institutes:grid.62560.37 schema:alternateName Department of Medicine, Section on Immunology and Immunogenetics, Joslin Diabetes Center, Brigham and Women's Hospital, Harvard Medical School, One Joslin Place, Boston, MA, USA
232 schema:name Department of Medicine, Section on Immunology and Immunogenetics, Joslin Diabetes Center, Brigham and Women's Hospital, Harvard Medical School, One Joslin Place, Boston, MA, USA
233 rdf:type schema:Organization
 




Preview window. Press ESC to close (or click here)


...