Chimeric EWSR1-FLI1 regulates the Ewing sarcoma susceptibility gene EGR2 via a GGAA microsatellite View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

2015-07-27

AUTHORS

Thomas G P Grünewald, Virginie Bernard, Pascale Gilardi-Hebenstreit, Virginie Raynal, Didier Surdez, Marie-Ming Aynaud, Olivier Mirabeau, Florencia Cidre-Aranaz, Franck Tirode, Sakina Zaidi, Gaëlle Perot, Anneliene H Jonker, Carlo Lucchesi, Marie-Cécile Le Deley, Odile Oberlin, Perrine Marec-Bérard, Amélie S Véron, Stephanie Reynaud, Eve Lapouble, Valentina Boeva, Thomas Rio Frio, Javier Alonso, Smita Bhatia, Gaëlle Pierron, Geraldine Cancel-Tassin, Olivier Cussenot, David G Cox, Lindsay M Morton, Mitchell J Machiela, Stephen J Chanock, Patrick Charnay, Olivier Delattre

ABSTRACT

Olivier Delattre and colleagues show that a Ewing sarcoma susceptibility variant at 10q21.3 influences EGR2 expression by altering the activity of an enhancer bound by EWSR1-FLI1. They further show that EGR2 knockdown inhibits growth of Ewing sarcoma cells in vitro and induces complete regression of xenografts in vivo, establishing a critical role for EGR2 in Ewing sarcomagenesis. More... »

PAGES

1073-1078

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  • Journal

    TITLE

    Nature Genetics

    ISSUE

    9

    VOLUME

    47

    Identifiers

    URI

    http://scigraph.springernature.com/pub.10.1038/ng.3363

    DOI

    http://dx.doi.org/10.1038/ng.3363

    DIMENSIONS

    https://app.dimensions.ai/details/publication/pub.1033290656

    PUBMED

    https://www.ncbi.nlm.nih.gov/pubmed/26214589


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