Tumor necrosis factor alpha and mesangial cells View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

1992-03

AUTHORS

Laurent Baud, Bruno Fouqueray, Carole Philippe, Abdelaziz Amrani

ABSTRACT

Bacterial infections promote important alterations in immune, hemodynamic, and metabolic function which can result in irreversible shock. These changes are mediated by host-secreted cytokines rather than by direct action of the endotoxin lipopolysaccharide (LPS) derived from the cell wall of bacteria. One cytokine that has been extensively studied for its role in infection is macrophage-derived tumor necrosis factor alpha (TNF) [1]. Three observations suggest that TNF has a central involvement in endotoxic shock: (i) High levels of TNF can be found in blood of animals and humans with endotoxic shock; (ii) When injected to animals, TNF causes a syndrome similar to endotoxic shock, with hypotension, decreased tissue perfusion, organ failure, and lactic acidosis; (iii) The effects of LPS can be prevented by prior passive immunization of animals with polyclonal or monoclonal antibody directed against TNF. More... »

PAGES

600-603

Identifiers

URI

http://scigraph.springernature.com/pub.10.1038/ki.1992.90

DOI

http://dx.doi.org/10.1038/ki.1992.90

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1000482721

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/1573835


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