Transgenic rescue of defective Cd36 ameliorates insulin resistance in spontaneously hypertensive rats View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2001-02

AUTHORS

Michal Pravenec, Vladimir Landa, Vaclav Zidek, Alena Musilova, Vladimir Kren, Ludmila Kazdova, Timothy J. Aitman, Anne M. Glazier, Azeddine Ibrahimi, Nada A. Abumrad, Nianning Qi, Jia-Ming Wang, Elizabeth M. St. Lezin, Theodore W. Kurtz

ABSTRACT

Spontaneously hypertensive rats (SHR) display several features of the human insulin-resistance syndromes. Cd36 deficiency is genetically linked to insulin resistance in SHR. We show that transgenic expression of Cd36 in SHR ameliorates insulin resistance and lowers serum fatty acids. Our results provide direct evidence that Cd36 deficiency can promote defective insulin action and disordered fatty-acid metabolism in spontaneous hypertension. More... »

PAGES

ng0201_156

References to SciGraph publications

Identifiers

URI

http://scigraph.springernature.com/pub.10.1038/84777

DOI

http://dx.doi.org/10.1038/84777

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1049119589

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/11175782


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