Telomerase prevents the accelerated cell ageing of Werner syndrome fibroblasts View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2000-01

AUTHORS

F S Wyllie, C J Jones, J W Skinner, M F Haughton, C Wallis, D Wynford-Thomas, R G Faragher, D Kipling

ABSTRACT

Werner syndrome (WS) is a rare disorder inherited in an autosomal recessive manner and characterized by accelerated ageing1. WS fibroblasts display an accelerated rate of senescence in vitro2, which has been linked to this progeroid phenotype. The senescence of normal human fibroblasts is triggered by telomere shortening3, 4, 5, whereas the premature senescence of WS fibroblasts has been assumed6, 7 to reflect the accumulation of DNA damage. Here we show that forced expression of telomerase in WS confers extended cellular lifespan and probable immortality. More... »

PAGES

16-17

References to SciGraph publications

Identifiers

URI

http://scigraph.springernature.com/pub.10.1038/71630

DOI

http://dx.doi.org/10.1038/71630

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1042917021

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/10615119


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