Mutations in the Plk gene lead to instability of Plk protein in human tumour cell lines View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2000-11

AUTHORS

Siro Simizu, Hiroyuki Osada

ABSTRACT

It has been established that mutations in Drosophila Polo cause abnormalities in mitosis. In human cells, maximal Plk activity is reached in the M phase of the cell cycle, and the function of Plk is therefore considered to be required for mitotic cellular events such as spindle formation, chromosome segregation and cytokinesis. Microinjection of anti-Plk antibody into living cells has been found to induce a mitotic abnormality that contributes to the generation of aneuploidy, and this is an important finding in relation to tumour development. Indeed, previous studies have shown that the level of expression of a mitotic checkpoint gene, hsMAD2, is reduced and that another checkpoint gene, BUB1, is mutated in certain human cancer cells. More... »

PAGES

ncb1100_852

References to SciGraph publications

Identifiers

URI

http://scigraph.springernature.com/pub.10.1038/35041102

DOI

http://dx.doi.org/10.1038/35041102

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1002882930

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/11056542


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