Mouse Models of Liver Fibrosis Mimic Human Liver Fibrosis of Different Etiologies View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

2014-12

AUTHORS

Allyson K. Martínez, Luca Maroni, Marco Marzioni, Syed T. Ahmed, Mena Milad, Debolina Ray, Gianfranco Alpini, Shannon S. Glaser

ABSTRACT

The liver has the amazing capacity to repair itself after injury; however, the same processes that are involved in liver regeneration after acute injury can cause serious consequences during chronic liver injury. In an effort to repair damage, activated hepatic stellate cells trigger a cascade of events that lead to deposition and accumulation of extracellular matrix components causing the progressive replacement of the liver parenchyma by scar tissue, thus resulting in fibrosis. Although fibrosis occurs as a result of many chronic liver diseases, the molecular mechanisms involved depend on the underlying etiology. Since studying liver fibrosis in human subjects is complicated by many factors, mouse models of liver fibrosis that mimic the human conditions fill this void. This review summarizes the general mouse models of liver fibrosis and mouse models that mimic specific human disease conditions that result in liver fibrosis. Additionally, recent progress that has been made in understanding the molecular mechanisms involved in the fibrogenic processes of each of the human disease conditions is highlighted. More... »

PAGES

143-153

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s40139-014-0050-2

DOI

http://dx.doi.org/10.1007/s40139-014-0050-2

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1023141836

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/25396098


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