A case of early recurrent immunoglobulin A nephropathy and T-cell-mediated rejection in a transplant patient with Wiskott–Aldrich syndrome View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

2021-07-25

AUTHORS

Kosei Yamaguchi, Mineaki Kitamura, Yuki Kawaguchi, Kanako Hayashi, Kumiko Muta, Masayuki Nakazawa, Tsuyoshi Matsuda, Toru Onita, Masaharu Nishikido, Hideki Sakai, Hiroshi Mukae, Tomoya Nishino

ABSTRACT

Wiskott–Aldrich syndrome (WAS) is an X-chromosome recessive immunodeficiency disease characterized by the triad of thrombocytopenia, eczema, and susceptibility to infection owing to WAS protein gene abnormalities. Kidney transplantation is rarely offered to WAS patients with end-stage renal disease because of concerns that thrombocytopenia and immune disorders may affect the clinical outcome. Here, we report the case of a 20-year-old kidney transplant patient who developed end-stage renal disease owing to immunoglobulin (Ig)A nephropathy caused by WAS. Despite recurrent IgA nephropathy and T-cell-mediated rejection 7 months after transplantation, two rounds of steroid pulse therapy attenuated his renal function and urinary abnormality. His serum creatinine level was maintained at approximately 1.5 mg/dL 1 year after transplantation. No other WAS-related complications were observed throughout the clinical course. Although WAS can cause poor prognosis in kidney transplant patients, careful follow-up may allow kidney transplantation to be performed. More... »

PAGES

60-66

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s13730-021-00631-9

DOI

http://dx.doi.org/10.1007/s13730-021-00631-9

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1139918661

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/34304384


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