The role of miRNA-155 in monocrotaline-induced pulmonary arterial hypertension through c-Fos/NLRP3/caspase-1 View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2020-05-25

AUTHORS

Shou-Dong Chai, Zhen-Kun Li, Rui Liu, Tao Liu, Ming-Feng Dong, Pei-Zhe Tang, Jian-Tang Wang, Sheng-Jun Ma

ABSTRACT

BackgroundNisoldipine can effectively suppress pulmonary arterial smooth muscle cell proliferation and c-Fos expression.ObjectiveTo identify the mechanism of the anti-inflammatory effects in monocrotaline-induced pulmonary arterial hypertension (PAH), focusing on the c-Fos/NLRP3/caspase-1 pathway.ResultsIn a mice model of monocrotaline-induced PAH, miRNA-155 expression was increased. In an in vitro model, overexpression of miRNA-155 promoted inflammation and induced c-Fos, NLRP3, and caspase-1 protein expression. The inhibition of c-Fos reduced the effects of miRNA-155 on inflammation in an in vitro model of monocrotaline-induced PAH. The inhibition of NLRP3 reduced the effects of miRNA-155 on inflammation in an in vitro model of monocrotaline-induced PAH.ConclusionsmiRNA-155 increased inflammation in monocrotaline-induced PAH through c-Fos/NLRP3/caspase-1. More... »

PAGES

311-320

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s13273-020-00083-9

DOI

http://dx.doi.org/10.1007/s13273-020-00083-9

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1127880153


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