Ongoing vascular inflammation evaluated by 18F-fluorodeoxyglucose positron emission tomography in patients long after Kawasaki disease View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2022-07-07

AUTHORS

Kenji Suda, Nobuhiro Tahara, Munehisa Bekki, Tomohisa Nakamura, Akihiro Honda, Shintaro Kishimoto, Yoshiyuki Kagiyama, Motofumi Iemura, Kiminori Fujimoto, Toshi Abe, Yoshihiro Fukumoto

ABSTRACT

BackgroundThis study aimed to determine whether ongoing vascular inflammation presents in patients who had coronary artery aneurysms (CAAs) caused by Kawasaki disease (KD).MethodsSubjects were 26 patients with a history of KD; 15 had giant CAA (gCAA) ≥ 8.0 mm and 11 had smaller CAA (smCAA) < 8 mm in the acute phase. They underwent X-ray computed tomography and 18F-fluorodeoxyglucose positron emission tomography. We determined the maximum coronary target-to-background ratio (CaTBR) and the mean thoracic aorta TBR (TaTBR) in each patient. They were compared between groups, and their correlation with various variables was determined.ResultsCaTBR and TaTBR were significantly higher in gCAA than in smCAA (P < .005 for both values) and were significantly higher even in patients without any metabolic risk factor (P < .05 for both values). The CAA size in acute phase significantly positively correlated with CaTBR (R2 = 0.32) as well as TaTBR (R2 = 0.28). Also, TaTBR significantly positively correlated with CaTBR (R2 = 0.32) as well as cumulative number of metabolic risk factors (trend, P = .03).ConclusionsOngoing vascular inflammation may present long after KD, especially in patients with severe inflammation expressed as gCAA in the acute phase. More... »

PAGES

1-12

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s12350-022-03041-1

DOI

http://dx.doi.org/10.1007/s12350-022-03041-1

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1149307347

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/35799038


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