Relationship between myocardial perfusion abnormalities and contractile impairment in anginal patients View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2014-12

AUTHORS

Alessia Gimelli, Riccardo Liga, Assuero Giorgetti, Mirta Casagranda, Paolo Marzullo

ABSTRACT

BACKGROUND: A relationship between left ventricular (LV) contractile impairment and myocardial perfusion abnormalities has been suggested. METHODS AND RESULTS: Three-hundred and thirty-seven patients underwent myocardial perfusion imaging at CZT and coronary angiography. On scintigraphic images, the summed difference score (SDS) and LV-ejection fraction (EF) were computed. Patients were categorized as follows: Group-1 (LV-EF < 40%; 71 patients), Group-2 (LV-EF ≥ 40% and < 55%; 77 patients), and Group-3 (LV-EF ≥ 55%; 189 patients). Significant coronary artery disease (CAD; ≥50% stenosis) was recognized in 159/337 (47%) patients. Interestingly, while in Group-3 subjects an inverse relationship between SDS values and post-stress LV-EF was evident (P < .001), Group-1 patients presented a significant association between an increased SDS and more elevated post-stress LV-EF values (P = .009). Similarly, despite in the overall population an increasing severity of CAD was associated with higher SDS values (P < .001), this relationship disappeared in Group-1 patients (P = .298). At multiple regression analysis, after correction for CAD, LV dysfunction was negatively associated with an elevated SDS (P = .018). Conversely in patients with normal LV function and no history of myocardial infarction, CAD extent, and functional measures of stress-induced myocardial ischemia were strictly correlated. CONCLUSIONS: Independently from CAD, a significantly impaired LV function associates with a lower prevalence of reversible ischemia. More... »

PAGES

1181-1190

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s12350-014-9950-0

DOI

http://dx.doi.org/10.1007/s12350-014-9950-0

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1044011104

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/25080198


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