Attachment-Related Mentalization Moderates the Relationship Between Psychopathic Traits and Proactive Aggression in Adolescence View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

2013-08

AUTHORS

Svenja Taubner, Lars O. White, Johannes Zimmermann, Peter Fonagy, Tobias Nolte

ABSTRACT

The lack of affective responsiveness to others' mental states - one of the hallmarks of psychopathy - is thought to give rise to increased interpersonal aggression. Recent models of psychopathy highlight deficits in attachment security that may, in turn, impede the development of relating to others in terms of mental states (mentalization). Here, we aimed to assess whether mentalization linked to attachment relationships may serve as a moderator for the relationship between interpersonal aggression and psychopathic traits in an adolescent community sample. Data from 104 males and females with a mean age of 16.4 years were collected on mentalization capacities using the Reflective Functioning Scale on the Adult Attachment Interview (AAI). Psychopathic traits and aggressive behavior were measured via self-report. Deficits in mentalization were significantly associated with both psychopathic traits and proactive aggression. As predicted, mentalization played a moderating role, such that individuals with increased psychopathic tendencies did not display increased proactive aggression when they had higher mentalizing capacities. Effects of mentalization on reactive aggression were fully accounted for by its shared variance with proactive aggression. Psychopathic traits alone only partially explain aggression in adolescence. Mentalization may serve as a protective factor to prevent the emergence of proactive aggression in spite of psychopathic traits and may provide a crucial target for intervention. More... »

PAGES

929-938

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s10802-013-9736-x

DOI

http://dx.doi.org/10.1007/s10802-013-9736-x

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1005139444

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/23512713


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