Eculizumab reversed severe distal ischemic syndrome and glomerulonephritis with isolated C3 deposits associated with anti-factor H autoantibodies: a case report View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2018-03-07

AUTHORS

Samuel Deshayes, Nicolas Martin Silva, Valérie Chatelet, Sylvain Chantepie, Moglie Le Quintrec, François Comoz, Frank Bridoux, Marie-Agnès Dragon-Durey, Achille Aouba

ABSTRACT

B-cell clones can produce a monoclonal immunoglobulin, which may be responsible for visceral involvements. Kidney involvement is frequent, affecting 20 to 50% of patients with multiple myeloma. One mechanism underlying this involvement is a dysregulation of the complement alternative pathway, leading to C3 glomerulopathies. We report a patient who had a multiple myeloma, C3 glomerulopathy related to factor H autoantibody, and digital ischemia, who was treated successfully with eculizumab, an anti-complement therapy, without any relapse in 2 years of follow-up. More... »

PAGES

1119-1122

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s10067-018-4058-6

DOI

http://dx.doi.org/10.1007/s10067-018-4058-6

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1101378161

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/29516279


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