Upper gastrointestinal ulcer in Japanese patients taking low-dose aspirin View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2009-02

AUTHORS

Akiko Shiotani, Takashi Sakakibara, Yoshiyuki Yamanaka, Hiroshi Imamura, Ken-Ichi Tarumi, Noriaki Manabe, Tomoari Kamada, Hiroaki Kusunoki, Jiro Hata, Ken Haruma

ABSTRACT

BACKGROUND: There are few studies on the association of the risks of upper gastrointestinal (GI) ulcer induced by aspirin combined with other medicines. We investigated the association between peptic ulcer and clinical parameters, including Helicobacter pylori infection and combinations of medicines. METHODS: Patients taking 100 mg aspirin for cardiovascular diseases who were planning to undergo endoscopy were enrolled. Serum H. pylori IgG antibody was measured. RESULTS: A total of 305 patients were enrolled, and 38 patients (12.4%) had ulcer lesions. Sex, smoking, drinking, body mass index, endoscopic findings for gastric atrophy (open type), or presence of H. pylori were not significantly associated with ulcer lesions. Cotreatment with anticoagulants [ticlopidine, 34.2% vs. 21.3%; adjusted odds ratio (OR), 3.1; 95% confidence interval (CI), 1.4-7.1; ticlopidine plus warfarin, 13.2% vs. 3.7%; adjusted OR, 4.4; 95% CI, 1.3-15], proton pump inhibitor (PPI 5.3% vs. 34.8%; adjusted OR, 0.10; 95% CI, 0.02-0.43), and antihypertensive medicine were significantly associated with peptic ulcer. Among antihypertensive medicines, AT1 receptor blocker and angiotensin-converting enzyme (ACE) inhibitor tended to be associated with upper GI ulcer. CONCLUSIONS: PPI was superior to H2-receptor antagonist for prevention of peptic ulcer, and cotreatment with AT1 receptor blocker or ACE inhibitor seemed to reduce peptic ulcer among patients taking low-dose aspirin. More... »

PAGES

126-131

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s00535-008-2290-6

DOI

http://dx.doi.org/10.1007/s00535-008-2290-6

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1038282328

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/19214674


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