LILRA6 copy number variation correlates with susceptibility to atopic dermatitis View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

2016-10

AUTHORS

M. R. López-Álvarez, W. Jiang, D. C. Jones, J. Jayaraman, C . Johnson, W. O. Cookson, M. F. Moffatt, J. Trowsdale, J. A. Traherne

ABSTRACT

Leukocyte immunoglobulin-like receptors (LILR) are expressed mostly on myelomonocytic cells where they are mediators of immunological tolerance. Two LILR genes, LILRA3 and LILRA6, exhibit marked copy number variation. We assessed the contribution of these genes to atopic dermatitis (AD) by analysing transmission in 378 AD families. The data indicated that copies of LILRA6 were over-transmitted to affected patients. They are consistent with a contribution of LILR genes to AD. They could affect the equilibrium between activating and inhibitory signals in the immune response. More... »

PAGES

743-747

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s00251-016-0924-z

DOI

http://dx.doi.org/10.1007/s00251-016-0924-z

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1013345470

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/27333811


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RDF/XML is a standard XML format for linked data.

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