Developmental perspectives of the drugs targeting enzyme-instigated inflammation: a mini review View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2019-04

AUTHORS

Parteek Prasher, Harish Mudila, Mousmee Sharma, Beena Khati

ABSTRACT

The instigation of chronic inflammation begins with the release of arachidonic acid (AA) from the membranous phospholipids through a biochemical reaction catalyzed by cytosolic phospholipase A2, which selectively hydrolyzes the arachidonyl phospholipid at the sn-2 position, thereby releasing a lysophospholipid and AA into the cytosol. This intracellular event swiftly progresses to the production of pro-inflammatory leukotrienes and prostaglandins by the catalytic action of the enzymes lipoxygenases and cyclooxygenases (COXs) on AA, respectively, whose anomalous production may manifest into chronic inflammation and related disorders. Despite several developments from aspirin to non-steroidal anti-inflammatory drugs and COX-2 inhibitors, the anti-inflammatory therapy, due to its associated side effects even in the advanced formulations, has suffered frequent setbacks and commercial withdrawals. The contemporary research therefore aims at designing novel candidate molecules with superior viability and an enhanced acceptability towards biological systems with a minimum conceivable side effect. More... »

PAGES

417-449

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    http://scigraph.springernature.com/pub.10.1007/s00044-019-02315-7

    DOI

    http://dx.doi.org/10.1007/s00044-019-02315-7

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    https://app.dimensions.ai/details/publication/pub.1112504358


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