Dysregulated glycolysis as an oncogenic event View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

2015-05

AUTHORS

Takumi Mikawa, Matilde E. LLeonart, Akifumi Takaori-Kondo, Nobuya Inagaki, Masayuki Yokode, Hiroshi Kondoh

ABSTRACT

Enhanced glycolysis in cancer, called the Warburg effect, is a well-known feature of cancer metabolism. Recent advances revealed that the Warburg effect is coupled to many other cancer properties, including adaptation to hypoxia and low nutrients, immortalisation, resistance to oxidative stress and apoptotic stimuli, and elevated biomass synthesis. These linkages are mediated by various oncogenic molecules and signals, such as c-Myc, p53, and the insulin/Ras pathway. Furthermore, several regulators of glycolysis have been recently identified as oncogene candidates, including the hypoxia-inducible factor pathway, sirtuins, adenosine monophosphate-activated kinase, glycolytic pyruvate kinase M2, phosphoglycerate mutase, and oncometabolites. The interplay between glycolysis and oncogenic events will be the focus of this review. More... »

PAGES

1881-1892

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/s00018-015-1840-3

DOI

http://dx.doi.org/10.1007/s00018-015-1840-3

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1045914411

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/25609364


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