Absence of H-2 genetic influence on streptozotocin-induced diabetes in mice View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

1982-08

AUTHORS

H. Kromann, M. Christy, J. Egeberg, Å. Lernmark, J. Nerup

ABSTRACT

Five daily injections of streptozotocin (40 mg/kg) produced a delayed but progressively increasing level of hyperglycaemia in long term studies with male Naval Medical Research Institute mice and C3D2F1 (DBA 2 J male x C3H/Tif female) F1 hybrid mice. The development of hyperglycaemia was paralleled by decreased amounts of pancreatic immunoreactive insulin as well as degranulation and necrosis of pancreatic B cells. Insulitis was found from days 9-25 after the first injection of streptozotocin. Compared with the F1 hybrid strain the parental inbred strains DBA 2 J and C3H/Tif demonstrated a certain resistance to streptozotocin. Development of hyperglycaemia did not differ in four congenic resistant lines of mice on the C57 BL/10 genetic background, indicating the major histocompatibility complex genes are not likely to determine susceptibility to streptozotocin-induced islet B cell damage. More... »

PAGES

114-118

References to SciGraph publications

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/bf01271171

DOI

http://dx.doi.org/10.1007/bf01271171

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1036830612

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/6215276


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