Metabolism of genetically obese rats on normal or high-fat diet View Full Text


Ontology type: schema:ScholarlyArticle      Open Access: True


Article Info

DATE

1974-11

AUTHORS

D. Lemonnier, R. Aubert, J. P. Suquet, G. Rosselin

ABSTRACT

Adult genetically obesefafa rats showed a high level of lipogenesis from glucose in liver but not in adipose tissue; pancreatic content and serum levels of insulin were elevated. Glucose uptake and insulin sensitivity were decreased in muscle,fafa rats and their lean littermates fed a high-fat diet showed increased fat deposits. Serum insulin levels were not significantly affected by diet in either group. The larger the fat cells were, the more actively they utilised glucose; insulin sensitivity was influenced both by diet and cell size. Control rats made obese by a high caloric diet did not show insulin resistance in muscle. — The data indicate that in adult obesity in these rats, even in the presence of marked hyperinsulinism, increased lipogenesis in adipose tissue is not a prerequisite. Rather, fat storage is a consequence of increased uptake of circulating triglycérides. On a diet rich in carbohydrate, adipose tissue fatty acids were mainly of hepatic origin; on a high-fat diet they were of dietary origin. More... »

PAGES

697-701

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/bf01222007

DOI

http://dx.doi.org/10.1007/bf01222007

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1046530866

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/4448331


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