Studien über die Folgen einer vorübergehenden experimentellen Nierenischämie View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

1963-07

AUTHORS

H. Cain, St. Fazekas

ABSTRACT

Vorübergehende experimentelle Ischämie (60 min) der Rattenniere hat weitreichende Strukturveränderungen an Arterien und Arteriolen sowie am Tubulusapparat zur Folge. Die ischämischen Gefäßwandläsionen erweisen sich alseine Ursache für eine allgemeine Zufluß-Störung. Sie können im postischämischen Stadium lokal gesteigert werden durch akzessorische temporäre Spasmen. Daraus resultieren überdauernde Blutverteilungsänderungen und eine Minderung der Filtration. Das glomeruläre Filter als solches erleidet nur einen leichten und rasch abklingenden Schaden (Permeabilitätserhöhung). Die Affektion der Tubuli bewirkt eine kräftige Ansammlung von Flüssigkeit in Tubuluszellen und im Tubuluslumen. Gleichzeitige Anreicherung des Primärharnes mit Eiweiß tubuloepithelialer Herkunft bedingt eine Viscositätserhöhung, die ihrerseits bedeutsamer Teilfaktor an der initialen Durchfluß-Störung in dem relativ zu engen Kanälchensystem ist. Der so erklärbare Anstieg des intratubulären Harndruckes kann zunächst nicht kompensiert werden durch konsekutive autoregulative Steigerung des intrarenalen Blutdruckes, da die hierfür verantwortlichen Regulatoren temporär hypoxisch geschädigt sind (= präglomeruläre Barriere als weitere Teilursache für die Oligo-Anurie). Somit führen bei der Ratte präglomeruläre und tubuläre Komponenten gemeinsam zur Filtrationsstörung und gleichzeitigen Durchflußbehinderung im Tubulussystem. More... »

PAGES

389-416

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    URI

    http://scigraph.springernature.com/pub.10.1007/bf00963736

    DOI

    http://dx.doi.org/10.1007/bf00963736

    DIMENSIONS

    https://app.dimensions.ai/details/publication/pub.1051425982

    PUBMED

    https://www.ncbi.nlm.nih.gov/pubmed/14017737


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