Blood affinity for oxygen in experimental hemorrhagic shock with metabolic acidosis View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

1975-03

AUTHORS

F. Lecompte, H. Aberkane, E. Azoulay, M. Muffat-Joly, J. J. Pocidalo

ABSTRACT

This study was designed to evaluate, in vivo, the effect of a severe non-respiratory acidosis on hemoglobin oxygen transport. Oxygen affinity of hemoglobin, Bohr effect, Hill's number and red cell 2,3-DPG were evaluated during experimental hemorrhagic shock in dogs. Three periods were considered: control, hypotension (mean arterial pressure 60 mm Hg for 2 hr 30 min) and blood replacement. There was no significant change in erythrocyte 2,3-DPG following hemorrhagic hypotension but ATP increased significantly. n, the Hill number (2.6), was not changed by in vivo acidosis (pH 7.1). Respiratory Bohr coefficient (BCO2) corresponding to pHe variations was drastically reduced (control BCO2 = 0.55, acidosis BCO2 = 0.31, blood replacement BCO2 = 0.35). P50(7.4) was not modified significantly by hemorrhagic acidosis. It is unlikely that variations of blood affinity for oxygen play a major role in oxygen delivery during early experimental hemorrhagic shock. More... »

PAGES

147-155

References to SciGraph publications

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/bf00581284

DOI

http://dx.doi.org/10.1007/bf00581284

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1027654465

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/1239727


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