The suppression of Haemoglobin E synthesis View Full Text


Ontology type: schema:ScholarlyArticle     


Article Info

DATE

1967-12

AUTHORS

S. Tuchinda, D. Beale, H. Lehmann

ABSTRACT

The blood of two infants with Haemoglobin E trait and a form of α-thalassaemia (Haemoglobin H Disease) was examined and it was confirmed that the proportion of Haemoglobin A:E was higher than in uncomplicated Haemoglobin E trait. Haemoglobin H (β4A) was added to the haemoglobin solution from a Haemoglobin E trait carrier. This mixture was dissociated into its α2, β2A and β2E subunits, and these were then recombined. The proportion of A:E had risen to that found in vivo in Haemoglobin E trait carriers with Haemoglobin H Disease. It is suggested that competition between βA and βE for α-chains may be an example of the mechanism by which α-thalassaemia ‘interacts’ with β-chain abnormal haemoglobins. More... »

PAGES

312-318

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/bf00292281

DOI

http://dx.doi.org/10.1007/bf00292281

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1042381949

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/6081911


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