Loss of Heterozygosity of the Long Arm of Chromosome 16 in Hepatocellular Carcinoma on Liver Cirrhosis View Full Text


Ontology type: schema:Chapter     


Chapter Info

DATE

1996

AUTHORS

Laura Gramantieri , Anna Casali , Stefano Gaiani , Fabio Piscaglia , Barbara Stecca , Alighiero Mazziotti , Antonino Cavallari , Luigi Bolondi

ABSTRACT

The analysis of loss of heterozygosity in human neoplasms, has shown that several genomic regions are frequently lost, suggesting their involvement in the pathogenesis of cancer as putative sites for tumor suppressor genes. The long arm of chromosome 16 has been implicated in breast, prostate and hepatocellular carcinomas. Previous studies made on large, advanced cases of HCC, revealed large areas of deletion on 16q. A recent report from a Japanese series of large HCC delimitated a common area of deletion at 16q22–23. We tested 16 small (<3 cm) HCC for loss of somatic heterozygosity using a set of 8 polymorphic microsatellite markers spanning 16q22–24. Three out of 10 informative cases showed loss of heterozygosity at 16q24.3, defining a commonly deleted region, possible site for a tumor suppressor gene. More... »

PAGES

174-182

References to SciGraph publications

Book

TITLE

New Trends in Hepatology

ISBN

978-94-010-6646-4
978-94-009-0357-9

Author Affiliations

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/978-94-009-0357-9_20

DOI

http://dx.doi.org/10.1007/978-94-009-0357-9_20

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1004893797


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