IgG4-Related Kidney Disease View Full Text


Ontology type: schema:Chapter      Open Access: True


Chapter Info

DATE

2013-10-12

AUTHORS

Takako Saeki , Mitsuhiro Kawano , Kazuhiro Yoshita , Mitsuhiro Ueno , Michio Nagata , Yutaka Yamaguchi

ABSTRACT

The main pathological finding in IgG4-related kidney disease (IgG4-RKD) is a form of tubulointerstitial nephritis (TIN), characterized by a dense lymphoplasmacytic infiltrate associated with abundant IgG4-positive plasma cells and fibrosis. Storiform fibrosis, the pattern of fibrosis often observed in IgG4-related TIN, is rarely associated with TIN of other causes, making it a useful differentiating feature. Other findings suggestive of IgG4-related TIN are as follows: clear demarcation between the affected and unaffected portions; extension of inflammatory cell infiltrates beyond the renal capsule; and eosinophil infiltration. By immunofluorescence and electron microscopy, immune complex deposits (or electron-dense deposits) are shown in tubular basement membrane in many cases. Although glomeruli generally show minor abnormalities, some glomerular lesions suggestive of immune complex involvement are also occasionally present. Among these glomerular lesions, membranous nephropathy and Henoch–Schönlein purpura nephritis have attracted the most attention. Further consideration must be given to these lesions as they may yield insights about the pathogenesis of IgG4-RKD. A goal of future work in IgG4-RKD is more extensive elucidation of the relationship between TIN and the glomerular lesions of IgG4-RKD. More... »

PAGES

169-179

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/978-4-431-54228-5_25

DOI

http://dx.doi.org/10.1007/978-4-431-54228-5_25

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1008499916


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