Nitric oxide, nitric oxide synthases and cyclooxygenase-2 in experimental focal stroke View Full Text


Ontology type: schema:Chapter     


Chapter Info

DATE

2001

AUTHORS

Xinkang Wang , Costantino Iadecola , Giora Z. Feuerstein

ABSTRACT

Nitric oxide (NO) is a unique messenger molecule involved in the regulation of diverse physiological processes including central and peripheral neurotransmission, smooth and cardiac muscle contractility, platelet aggregation, and immune cell functions. NO is crucial for many physiological functions; however, inappropriate release of this mediator is associated with a number of pathological conditions [1]. NO is synthesized from a guanidino group (by oxidation) of L-arginine by the enzyme NO synthase (NOS) in virtually all mammalian cells, including endothelial cells, neurons, glia, and cells of the immune system [2] (Fig. 1). Three isoforms of NOS have been characterized, including neuronal NOS (nNOS or type I), inducible or immunological NOS (iNOS or type II), and endothelial NOS (eNOS or type III). eNOS and nNOS are constitutively expressed. In contrast, iNOS is not normally expressed but its expression can be induced in many cell types by selected immunological stimuli [2]. The constitutive expression of eNOS and nNOS contributes to small but consistent amounts of NO production that are sufficient for signaling function. A massive induction of iNOS results in the production of large amounts of NO that may result in pathophysiological consequences [3]. More... »

PAGES

343-350

References to SciGraph publications

Book

TITLE

Inflammation and Stroke

ISBN

978-3-0348-9508-8
978-3-0348-8297-2

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/978-3-0348-8297-2_26

DOI

http://dx.doi.org/10.1007/978-3-0348-8297-2_26

DIMENSIONS

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