Acetaldehyde-induced Barrier Disruption and Paracellular Permeability in Caco-2 Cell Monolayer View Full Text


Ontology type: schema:Chapter     


Chapter Info

DATE

2008

AUTHORS

R K Rao

ABSTRACT

A significant body of evidence indicates that endotoxemia plays a crucial role in the pathogenesis of alcoholic liver disease. There are several possible factors that may be involved in inducing alcoholic endotoxemia, but increased intestinal permeability to enteric endotoxins appears to be the major contributing factor. In the normal gut, the epithelial barrier function prevents diffusion of toxins across the epithelium. However, the barrier is disrupted in patients with alcoholic liver disease. We showed that acetaldehyde disrupts intestinal epithelial tight junctions and increases paracellular permeability to endotoxins in Caco-2 cell monolayer, the extensively studied model of the differentiated intestinal epithelium. The mechanisms involved in acetaldehyde-induced increase in intestinal permeability to endotoxins can be elucidated in this model of the intestinal epithelium. More... »

PAGES

171-183

References to SciGraph publications

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/978-1-59745-242-7_13

DOI

http://dx.doi.org/10.1007/978-1-59745-242-7_13

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1027412293

PUBMED

https://www.ncbi.nlm.nih.gov/pubmed/18369919


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