Progressive Factors in Oncogene Transfected Rodent Embryo Fibroblasts View Full Text


Ontology type: schema:Chapter     


Chapter Info

DATE

1991

AUTHORS

Heather Marshall , Przemyslaw Popowicz , Georg Engel , Catharina Svensson , Göran Akusjärvi , Stig Linder

ABSTRACT

Activated ras oncogenes are inefficient in the transformation of primary rodent embryo fibroblasts (REFs). However, in unison with viral or cellular genes such as the adenovirus E1A, polyoma large-T, mutant p53 or myc genes, transformation will occur. No common biochemical activity has been described for these cooperating oncogenes. We and others have demonstrated that treatment of T24-ras oncogene transfected REF cells with the glucocorticoid dexamethasone (DEX) facilitates transformation (Martens et al., 1988; Yamashita et al., 1988; Marshall et al., Exp. Cell. Res, in press;). During a critical period between 1–3 months after transfection, cellular growth was found to be dependent on glucocorticoid. Hormone independence invariably develops during subsequent culture. These observations raise the possibility of hormonal promotion and progression of rat embryo fibroblasts expressing activated ras genes, and may offer a convenient model for studies of promotion and progression in vitro. More... »

PAGES

111-119

Book

TITLE

The Superfamily of ras-Related Genes

ISBN

978-1-4684-6020-9
978-1-4684-6018-6

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/978-1-4684-6018-6_13

DOI

http://dx.doi.org/10.1007/978-1-4684-6018-6_13

DIMENSIONS

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