NO and Glomerulonephritis View Full Text


Ontology type: schema:Chapter     


Chapter Info

DATE

1997

AUTHORS

Victoria Cattell , H. Terence Cook

ABSTRACT

The various forms of human glomerulonephritis are a group of diseases of largely unknown etiology, which account for a significant proportion of human renal disease. Most have an immune pathogenesis and injury results from either immune complex deposition or delayed hypersensitivity-type reactions within the glomerulus [1]. In the majority of cases, evidence from animal models suggests that immune complexes form in situ (deposition of circulating preformed immune complexes is now considered a rare event, with the possible exception of acute serum sickness). In these in situ reactions, the antigens may be fixed intrinsic epitopes on glomerular cells or matrix molecules (e.g., the Heymann antigen on glomerular visceral epithelial cells, or the Goodpasture antigen in the glomerular basement membrane), or they can be exogenous antigens which localize due to some particular affinity for glomerular structures (e.g., components of bacteria). In the former case, the corresponding autoantibodies form through the breakdown of self-tolerance. Recently, it has been appreciated through the development of new experimental models that some types of glomerulonephritis may arise without soluble antibody formation and have characteristics of type IV immune reactions, involving sensitized T lymphocytes. More... »

PAGES

307-331

References to SciGraph publications

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  • Book

    TITLE

    Nitric Oxide and the Kidney

    ISBN

    978-1-4613-7768-9
    978-1-4615-6039-5

    Identifiers

    URI

    http://scigraph.springernature.com/pub.10.1007/978-1-4615-6039-5_15

    DOI

    http://dx.doi.org/10.1007/978-1-4615-6039-5_15

    DIMENSIONS

    https://app.dimensions.ai/details/publication/pub.1034392358


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