The Humoral Anti-Islet Immune Response: Cytoplasmic Islet-Cell Antibodies: Technical Aspects and Clinical Applications View Full Text


Ontology type: schema:Chapter     


Chapter Info

DATE

1990

AUTHORS

Helga Gleichmann , Gian Franco Bottazzo

ABSTRACT

For many decades, insulin dependent diabetes mellitus (IDDM, type I diabetes) was regarded as a metabolic disorder with a typical acute clinical onset. In the last decade, however, a number of studies have led to substantial changes in previous concepts of the etiopathogenesis of the disease. Immunologic, genetic, and epidemiologic studies (for review, see Refs. 1–3) indicate that the majority of patients with IDDM have a long, clinically asymptomatic, prediabetic period. This phase is characterized by autoimmune phenomena and very subtle metabolic changes,4 which may antedate the clinical manifestation of insulin deficiency for many months and sometimes years. Organ-specific autoimmune reactions most likely play an essential role in the progressive destruction of the insulin-producing beta-cells of the islets of Langerhans; unequivocal proof for a direct autoimmune attack against beta-cells has yet to be demonstrated, however, despite the finding of a constellation of immune abnormalities around and inside injured islets at the time of diagnosis.5 More... »

PAGES

68-86

Book

TITLE

Autoimmunity and the Pathogenesis of Diabetes

ISBN

978-1-4612-7920-4
978-1-4612-3218-6

Identifiers

URI

http://scigraph.springernature.com/pub.10.1007/978-1-4612-3218-6_4

DOI

http://dx.doi.org/10.1007/978-1-4612-3218-6_4

DIMENSIONS

https://app.dimensions.ai/details/publication/pub.1007974390


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