Akihiko Takashima

Ontology type: schema:Person     

Person Info





Publications in SciGraph latest 50 shown

  • 2017-12 Mutation-induced loss of APP function causes GABAergic depletion in recessive familial Alzheimer’s disease: analysis of Osaka mutation-knockin mice in ACTA NEUROPATHOLOGICA COMMUNICATIONS
  • 2017-12 Treatment of intermittent hypoxia increases phosphorylated tau in the hippocampus via biological processes common to aging in MOLECULAR BRAIN
  • 2017-09-05 Polyunsaturated fatty acid deficiency during neurodevelopment in mice models the prodromal state of schizophrenia through epigenetic changes in nuclear receptor genes in TRANSLATIONAL PSYCHIATRY
  • 2016-12 Bexarotene targets autophagy and is protective against thromboembolic stroke in aged mice with tauopathy in SCIENTIFIC REPORTS
  • 2015-12 Toxic tau oligomer formation blocked by capping of cysteine residues with 1,2-dihydroxybenzene groups in NATURE COMMUNICATIONS
  • 2015 Toxic Tau Aggregation in AD in GENEDIS 2014
  • 2014-05 Neurofibrillary tangle formation by introducing wild-type human tau into APP transgenic mice in ACTA NEUROPATHOLOGICA
  • 2010-12-02 Tauopathy and Brain Aging in CYTOSKELETON OF THE NERVOUS SYSTEM
  • 2010-10-22 Presenilin-Based Transgenic Models of Alzheimer’s Dementia in ANIMAL MODELS OF DEMENTIA
  • 2007-09 Pore-Forming Proteins Share Structural and Functional Homology with Amyloid Oligomers in NEUROMOLECULAR MEDICINE
  • 2006-07 The players on the γ-secretase team in NATURE MEDICINE
  • 2002 Synthesis of the His 6-Tagged Recombinant Protein APPC99-His and His-PS1 Using RTS 500 in CELL-FREE TRANSLATION SYSTEMS
  • 2002 Production and Analysis of Human Mutant Tau (V337M) Transgenic Mouse in MAPPING THE PROGRESS OF ALZHEIMER’S AND PARKINSON’S DISEASE
  • 2001 Mechanism of neuron death in Alzheimer’s disease in NEUROSCIENTIFIC BASIS OF DEMENTIA
  • 1999-12 Presenilin-1 mutations downregulate the signalling pathway of the unfolded-protein response in NATURE CELL BIOLOGY
  • 1996-08 Preferential labeling of Alzheimer neurofibrillary tangles with antisera for tau protein kinase (TPK) I/glycogen synthase kinase-3β and cyclin-dependent kinase 5, a component of TPK II in ACTA NEUROPATHOLOGICA
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